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h after infectious diseases in low- and middle-income countries . In those surviving the immediate lethal injury, the most important cause of death is hypovolemic circulatory collapse (HCC) . The proposed mechanisms for development of HCC are cardiac dysfunction, abnormalities in vascular tone and capillary leak [4,5]. Better delineation of the specific contributions of each of these abnorm
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cycle from the G1 towards the S phase is controlled by the activities of CDKs and their partners, cyclins. E-type cyclins are G1 cyclins and have already been thought to be expected for the transition from the G1 for the S phase . Nonetheless, they may be dispensable for standard mitotic cell division inside the mouse, provided that mice deficient in each cyclin E1 and E2 create pretty much u